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Natural herbal &
homeopathic help for people suffering from Gout
Gout
HEALTH HERBS & NUTRITION
- REMEDIES - BONES, JOINTS AND MUSCLES - GOUT

About Uric Acid
Uric acid is a substance that results from the breakdown of purines (colorless crystalline organic bases
containing nitrogen).
A normal part of all human tissue, purines are found in many foods.
Normally, uric acid is dissolved in the blood and passed through the
kidneys into the urine, where it is eliminated.
If there is an increase in the production of uric acid or if the
kidneys do not eliminate enough uric acid from the body, levels of
it build up in the blood (a condition called hyperuricemia).
Hyperuricemia also may result when a person eats too many high-purine
foods, such as liver, dried beans and peas, anchovies, and gravies.
Hyperuricemia is not a disease, and by itself it is not dangerous.
However, if excess uric acid crystals form as a result of hyperuricemia, gout can develop. The crystals form and accumulate in
the joint, causing inflammation.
About Gout
Gout is a painful condition that occurs when the bodily waste
product uric acid is deposited as needle-like crystals in the joints
and/or soft tissues. In the joints, these uric acid crystals cause
inflammatory arthritis, which in turn leads to intermittent
swelling, redness, heat, pain, and stiffness in the joints.
In many people, gout initially affects the joints of the big toe (a
condition called podagra). But many other joints and areas around
the joints can be affected in addition to or instead of the big toe.
These include the insteps, ankles, heels, knees, wrists, fingers,
and elbows. Chalky deposits of uric acid, also known as tophi, can
appear as lumps under the skin that surrounds the joints and covers
the rim of the ear. Uric acid crystals can also collect in the
kidneys and cause kidney stones.
The Four Stages of Gout
Literally translated, arthritis means “joint inflammation.” It
refers to more than 100 different diseases that affect the joints. Gout accounts for approximately 5 percent of
all cases of arthritis.
The disease can progress through four stages:
Asymptomatic (without symptoms) Hyperuricemia: In this
stage, a person has elevated levels of uric acid in the blood (hyperuricemia),
but no other symptoms. Treatment is usually not required.
Acute Gout or Acute Gouty Arthritis: In this stage, hyperuricemia has caused the deposit of uric acid crystals in joint
spaces. This leads to a sudden onset of intense pain and swelling in
the joints, which also may be warm and very tender. An acute attack
commonly occurs at night and can be triggered by stressful events,
alcohol or drugs, or the presence of another illness. Attacks
usually subside within 3 to 10 days, even without treatment, and the
next attack may not occur for months or even years. Over time,
however, attacks can last longer and occur more frequently.
Interval or
Intercritical Gout: This is the period between
acute attacks. In this stage, a person does not have any symptoms.
Chronic
Tophaceous Gout: This is the most disabling stage
of gout. It usually develops over a long period, such as 10 years.
In this stage, the disease may have caused permanent damage to the
affected joints and sometimes to the kidneys. With proper treatment,
most people with gout do not progress to this advanced stage.
Gout is sometimes confused with other forms of arthritis because the
symptoms – acute and episodic attacks of joint warmth, pain,
swelling, and stiffness – can be similar. One form of arthritis
often confused with gout is called pseudogout. The pain, swelling,
and redness of pseudogout can also come on suddenly and may be
severe, closely resembling the symptoms of gout. However, the
crystals that irritate the joint are calcium phosphate crystals, not
uric acid. Therefore, pseudogout is treated somewhat differently and
is not reviewed in this booklet.


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Hyperuricemia |
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Presence of uric acid crystals in joint fluid |
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More than one attack of acute arthritis |
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Arthritis that develops in a day, producing a swollen,
red, and warm joint |
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Attack of arthritis in only one joint, often the toe,
ankle, or knee
Gout may be difficult for doctors to diagnose because
the symptoms can be vague, and gout often mimics other
conditions. Although most people with gout have hyperuricemia at some time during the course of their
disease, it may not be present during an acute attack.
In addition, having hyperuricemia alone does not mean
that a person will get gout. In fact, most people with hyperuricemia do not develop the disease.
To confirm a diagnosis of gout, a doctor may insert a
needle into an inflamed joint and draw a sample of synovial fluid, the substance that lubricates a joint.
The joint fluid is placed on a slide and examined under
a microscope for uric acid crystals. Their absence,
however, does not completely rule out the diagnosis.
The doctor also may find it helpful to look for uric
acid crystals around joints to diagnose gout. Gout
attacks may mimic joint infections, and a doctor who
suspects a joint infection (rather than gout) may also
culture the joint fluid to see whether bacteria are
present. |

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A number of risk factors are associated with hyperuricemia and gout.
They include: |

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Genetics - Twenty percent of people with gout have a
family history of the disease. |
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Gender and age - It is more common in men than in women
and more common in adults than in children. |
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Alcohol consumption -
Drinking too much alcohol can lead
to hyperuricemia, because alcohol interferes with the
removal of uric acid from the body. |
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Diet - Eating too many foods that are rich in purines
can cause or aggravate gout in some people. |
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Lead exposure - In some cases, exposure to lead in the
environment can cause gout. |
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Renal insufficiency, or the
inability of the kidneys to eliminate waste products, is
a common cause of gout in older people. |
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Other medical
problems that contribute to high blood levels of uric
acid include:
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high blood pressure
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hypothyroidism (underactive thyroid gland)
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conditions that cause an excessively rapid turnover of
cells, such as psoriasis,
hemolytic anemia, or some
cancers
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Kelley-Seegmiller syndrome or Lesch-Nyhan syndrome,
two rare conditions in which the enzyme that helps
control uric acid levels either is not present or is
found in insufficient quantities. |
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Medications - A number of medications may put people at
risk for developing hyperuricemia and gout. They
include:
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Diuretics, such as furosemide (Lasix),
hydrochlorothiazide (Esidrix, Hydro-chlor), and
metolazone (Diulo, Zaroxolyn)which are taken to
eliminate excess fluid from the body in conditions like
hypertension, edema, and heart disease, and which
decrease the amount of uric acid passed in the urine
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Salicylate-containing drugs, such as aspirin
›Niacin, a vitamin also known as nicotinic acid
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Cyclosporine (Sandimmune, Neoral), a medication that
suppresses the body’s immune system (the system that
protects the body from infection and disease). This
medication is used in the treatment of some autoimmune
diseases, and to prevent the body’s rejection of
transplanted organs.
›Levodopa (Larodopa), a medicine used to support
communication along nerve pathways in the treatment of
Parkinson’s disease. |

Pain relievers such as Tylenol or other more potent analgesics are used to
manage pain. Anti-inflammatory agents such as nonsteroidal anti-inflammatory
drugs (NSAIDS) are used to decrease joint inflammation, but common side
effects of NSAIDS include irritation of the gastrointestinal system, stomach
ulcers and intestinal problems, and even intestinal bleeding.
Corticosteroids, such as prednisone, given in short courses, are powerful
anti-inflammatory agents for treating acute gout; yet long-term chronic use of
corticosteroids is discouraged because of serious long-term side effects.
Probenecid (Benemid) and sulfinpyrazone (Anturane) are medications that are
commonly used to decrease uric acid blood levels by increasing the excretion of
uric acid into the urine. Since these drugs can cause kidney stones patients
with a history of kidney stones should seek other options. Allopurinol (Zyloprim)
lowers the blood uric acid level by preventing uric acid production, but
medication is used with caution in patients with poor kidney function, as they
are at a particular risk of developing side effects, including rash and liver
damage.
With
proper treatment, most people who have gout are able to control
their symptoms and live productive lives. Gout can be treated with
one or a combination of therapies. The goals of treatment are to
ease the pain associated with acute attacks, to prevent future
attacks, and to avoid the formation of tophi and kidney stones.
Successful treatment can reduce discomfort caused by the symptoms of
gout, as well as long-term damage to the affected joints. Treatment
will help to prevent disability due to gout.
Herbal
and homeopathic remedies have been used for centuries to treat a
number of conditions, including gout.
Homeopathic
ingredients like
Antimonium crud,
Quercus, Rhododendron, Benzoic ac, Silicea (6X), Nux vom & Ledum relieve
gout symptoms, including swelling, inflammation and burning pain in small
joints, especially the big toe.
Harpagophytum procumbens ('Sengaparile,' 'Devil's
Claw' or 'Duiwelsklou') (indigenous to the Kalahari Desert and is exclusive to Africa) has
been used for many years to promote joint health and stability. Glucosamine sulphate has been approved for the promotion of joint
and cartilage health in more than 70 countries throughout the world,
while Boswellia serata extracts address swelling joints, pain and
tender tissues naturally, without side effects.
Together, these
herbal + homeopathic remedies can:
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Relieve gout
symptoms
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Relieve acute
burning pains in joints
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Relieve swelling
and inflammation, especially in toes and small joints
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Assist the body
in reducing uric acid build up in joints
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Are safe for all
ages, including pregnant women or those who may be breastfeeding.
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Maintain
healthy, mobile joints and muscles
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Keep all joints
moving freely
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Support health
in large joints as well as small joints of the hands, feet, toes, elbows and
knees
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Maintain healthy
cartilage and connective tissue
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Maintain comfort
levels in all joints
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Gout-Gone™
Natural remedy helps lower uric acid for relief of swollen joints and pain in the toes and foot.

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JointEase Plus
Promotes joint and muscular-skeletal
system health.

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It is usually recommended that you lose weight (to those
who are overweight); limiting alcohol consumption; and
avoiding or limiting high-purine foods, which can
increase uric acid levels. |
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Drink plenty of nonalcoholic fluids, especially water. Nonalcoholic fluids help remove uric acid from the body.
Alcohol, on the other hand, can raise the levels of uric
acid in your blood. |
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Drinking ordinary black tea can help ward off an
attack of gout, possibly due to flavones it contains,
which inhibit xanthine oxidase, an enzyme that is
involved in the synthesis of uric acid. |
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Avoid foods that are high in purines such as anchovies,
asparagus, beef kidneys, brains, dried beans and peas,
game meats, gravy, herring, liver, mackerel, mushrooms,
sardines, scallops and sweetbreads. |
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Other foods to avoid include refined carbohydrates such as white flour; oats; yeasty foods such as beer and baked goods and vegetables like cauliflower and
spinach. |
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Vitamin C, taken in high doses, can help decrease blood
uric acid levels, but should not be taken without a
doctor's supervision. Note that there is a small subset
of people with gout who will actually get worse with
high levels of vitamin C. Also, a single high does can
free up too much uric acid and cause kidney stones. |


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During an acute attack, try to stay off your feet as much as possible and keep the affected joint
elevated. This actually will not be a big problem as an
acute attack generally makes the affected joint so
tender that it cannot even bear the load of a leaf! |
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If you can stand it, apply an icepack for 20
minutes or so. The cold will dull the pain and bring
down the swelling. Wrap the ice in a cloth to protect
the skin. Do this thrice a day. |
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If you take diuretics (such as for high blood
pressure), ask your doctor about the alternatives.
Diuretics eliminate excess fluids from the body; as a
side effect they reduce the amount of uric acid that
passes in the urine. less passing means more will remain
inside the body which will eventually worsen your gout. |
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Exercise regularly and maintain a healthy body weight.
Lose weight if you are overweight, but avoid
low-carbohydrate diets that are designed for quick
weight loss. When carbohydrate intake is insufficient,
your body can’t completely burn its own fat. As a
consequence, substances called ketones form and are
released into the bloodstream, resulting in a condition
called ketosis. After a short time, ketosis can increase
the level of uric acid in your blood. |
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Hot and cold compresses - Three minutes hot alternated
with 30 seconds cold provide pain relief and increase
circulation. |
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Nettle tea compress, applied externally. Use 1 -
2 tsp. per cup of hot water. Soak a clean cloth in this
tea and apply it to the tender joint. |
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Gout-Gone™
Natural remedy helps lower uric acid for relief of swollen joints and pain in the toes and foot.

|

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JointEase Plus
Promotes joint and muscular-skeletal
system health.

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